Bleeding is described as blood flow from blood vessels, and may be internal or external. Minor bleeding is seldom serious, as in the case of a common nosebleed or menstruation.
In severe cases, as when an artery is torn or severed, blood may flow so rapidly a blood clot cannot form. Normally the damaged blood vessels contract, slowing the flow of blood and thus allowing a clot to form, which plugs the wound. The vessels can then heal.
Severe bleeding is said to have occurred if one-and-a-half pints or more of blood are lost from an adult, or more than a half pint from a child. This can lead to shock and, ultimately, death in a very short period of time. Therefore, the emergency procedure to stop blood loss takes priority over any other problem.
Emergency first aid for external bleeding requires the person lie down, with affected part raised, to slow down blood flow and forestall shock. Easily reached and visible debris should be removed from the wound. Do not remove anything which is deeply embedded. The priority is to stop the bleeding. Infection prevention is secondary.
Use the cleanest available pad (a towel, a piece of clothing) or, if no material is available, use the hands and press down firmly on the wound, holding the edges together if they separate. Direct pressure around any embedded objects rather than over them. For uncontrollable bleeding, apply pressure on the major artery between the wound and heart.
When the bleeding stops, tie the pad firmly over the wound so as to maintain pressure. Never remove the pad if bleeding recurs. Should bleeding recur, tie additional padding over any padding already in place.
Only a doctor should remove the dressing. Tourniquets should not be applied except in life-threatening situations. Tourniquets tend to do more damage than the original injury, including loss of the extremity. Anticoagulant drugs should not be given the individual.
However slight, bleeding from any body orifice may indicate internal problems. Keep the victim warm, motionless, and loosen tightly-bound clothing.
Do not give food or fluids: if emergency treatment is required, digestion will interfere with the use of anesthetics.
Follow the treatment above and seek medical attention immediately.
External and Internal
Deficiency in any blood clot factor, especially blood platelet count.
Vitamin K deficiency which may be due to:
Inability of intestinal wall to absorb fats, vitamin K being a fat-soluble vitamin
Liver diseases, which suppress the formation of prothrombin and other blood clotting factors, or which prevent the formation of bile which aids vitamin K absorption, including:
Acute yellow atrophy
Low blood calcium
Iron deficiency anemia
Low dietary vitamin C causing defective blood platelet formation and is involved in the integrity of small blood vessel walls.
Nosebleeds may be caused by:
Colds which produce crusting and damage to membranes
Broken bones (without breakage of skin)
Use of oral contraceptives
Aspirin, which may cause gastrointestinal bleeding
Pregnancy; bleeding at this time may indicate a serious complication
Signs & Symptoms
Flow of blood from any area
Internal (Primarily the signs of incipient shock)
Weak or rapid pulse Cold skin Clammy skin Thirst Anxiety Nausea Vomiting Bruises Acute pain Dilated pupils which respond slowly to light
Structure & Function: Circulatory Support
Adult Child/Adolescent Acidophilus 2 - 3 tsp 1 - 2 tsp Calcium 400 - 800 mg 200 - 400 mg Quercetin* Vitamin E 100 - 400 IU 50 - 200 IU Vitamin K 1 - 5 mcg 0.5 - 2 mcg
* Please refer to the respective topic for specific nutrient amounts.
Note: All amounts are in addition to those supplements having a Recommended Dietary Allowance (RDA). Due to individual needs, one must always be aware of a possible undetermined effect when taking nutritional supplements. If any disturbances from the use of a particular supplement should occur, stop its use immediately and seek the care of a qualified health care professional.
The blood clotting time and the fragility of blood vessels determine how much bleeding results from trauma or illness.
Prolonged blood clotting time can be due to vitamin K deficiency. This can be remedied through vitamin K supplements or by increasing the dietary intake of the following items in a Dietary Goals Diet:
Beef liver Dark green leafy vegetables Cauliflower Green peas Cereals Chicken liver Egg yolk Milk Veal liver
Vitamin A toxicity may also decrease blood clotting time. Toxicity is associated with vitamin A doses greater than 50,000 IU per day.
Capillary fragility can be an indication of calcium deficiency. The integrity of blood vessels can be improved with calcium supplementation or by eating calcium-rich foods.
Vitamin C deficiency can also cause capillary fragility. It is manifested by a tendency to bruise, bleeding of the joints and gums, and petechial or pinpoint hemorrhages. Scurvy is the clinical condition for vitamin C deficiency in its extreme form. Scurvy is rare in the United States. Vitamin C supplementation or the addition of vitamin-rich foods to the diet will reverse the symptoms of vitamin C deficiency.
Anticoagulant properties of coumarins are increased if it is used in conjunction with salicylates such as aspirin or acidic sulfonamides. A tendency to bleed will occur. Gastric bleeding may be due to the usage of steroids. This medication should be taken with meals or, perhaps with antacids.
1.** Crotalus horridus - 6C to 15C or higher, depending on severity
2.* Phosphorus - 15C or higher
3.* Lachesis mutus tinct. - 30C once per day and 2X per per week for one month
4.** Ipecacuanha - 30C
5.***Ficus religiosa - 3X
6.***Cinnamomum - 3X to 30C probably the best, single remedy. Do not use with aconite
Doses cited are to be administered on a 3X daily schedule, unless otherwise indicated. Dose usually continued for 2 weeks. Liquid preparations usually use 8-10 drops per dose. Solid preps are usually 3 pellets per dose. Children use 1/2 dose.
X = 1 to 10 dilution - weak (triturition)
C = 1 to 100 dilution - weak (potency)
M = 1 to 1 million dilution (very strong)
X or C underlined means it is most useful potency
Asterisk (*) = Primary remedy. Means most necessary remedy. There may be more than one remedy - if so, use all of them.
Boericke, D.E., 1988. Homeopathic Materia Medica.
Coulter, C.R., 1986. Portraits of Homeopathic Medicines.
Kent, J.T., 1989. Repertory of the Homeopathic Materia Medica.
Koehler, G., 1989. Handbook of Homeopathy.
Shingale, J.N., 1992. Bedside Prescriber.
Smith, Trevor, 1989. Homeopathic Medicine.
Ullman, Dana, 1991. The One Minute (or so) Healer.
Note: The misdirected use of an herb can produce severely adverse effects, especially in combination with prescription drugs. This Herbal information is for educational purposes and is not intended as a replacement for medical advice.
Coagulant components Effect
Agrimony Increases human blood coagulation up to 50% Goldenseal Heparin antagonist Mistletoe Lectins, agglutinating activity Yarrow Coagulant, in vivo
It is also necessary to be aware of herbs which may have an anticoagulant effect and, therefore, potentiate medication.
Anticoagulant Components Effect
Alfalfa 2 Coumarin constituents Angelica 2 Coumarin constituents Angelica 39, 44, 45 Inhibits platelet aggregation Anise /Aniseed 2 Coumarin constituents Arnica 2 Coumarin constituents Asafetida 2 Coumarin constituents Anticoagulant in vivo Bogbean 2 Hemolytic activity Celery seed or extract 2 Coumarin constituents Cassia 2 Inhibits platelet aggregation Chamomile, German 2 Coumarin constituents Chamomile, Roman 2 Coumarin constituents Clove 2 Eugenol powerful inhibitor of platelet activity Cumin/Turmeric 34 Inhibits platelet aggregation Fenugreek 2 Coumarin constituents Feverfew 3 Inhibits platelet aggregation in vitro Fucus 2 Anticoagulant action Garlic 2, 7-16, 47 Interaction with warfarin reported Ganoderma 36, 37, 45 Inhibits platelet aggregation Ginger 2, 16-18 Inhibition of platelet activity Ginkgo 4 Inhibits platelet aggregation Ginseng 2, 5, 19-22, 45 Reduction of blood coagulation2/ Decreases INR5 Horse chestnut Coumarin constituents Horse radish Peroxidase stimulates synthesis of Arachidonic acid metabolites Isatis 38 Inhibits platelet aggregation Licorice Inhibition of platelet activity Meadowsweet Salicylate constituents Melilot 6 Coumarin constituents Methyl salicylate oil 26 Inhibits platelet aggregation Pau d'arco (Taheebo, Lapacho) 41, 48 Coumarin constituents Poplar Salicylate constituents Prickly Ash Coumarin constituents Quassia Coumarin constituents Red clover 2, 42 Coumarin constituents Salvia 29, 40, 43, 45 Inhibits platelet aggregation Shitake 36 Inhibits platelet aggregation Sweet Woodruff 6 Coumarin constituents Tanacetum 31-33 Anticoagulant Tonka beans 6 Coumarin constituents Tremella 36 Inhibits platelet aggregation Danshen 26, 27 Anticoagulant Breviscopine 28 Anticoagulant Umbelliferae 30 Coumarin constituents Willow Salicylate constituents
These numbers refer to their respective references:
1. Coumadin Package insert.
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4. Ginko Monograph. In: Burnhan TH, Hagemann RC, Threlkeld DS (eds.) The Lawrence Review of Natural Products. St. Louis: Facts and Comparisons. 1994.
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6. Ridker PM. Toxic effects of herbal teas. Arch Environ Health 1987;42(2):133-6.
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21. Greenspan EM. Ginseng and vaginal bleeding. Letter to the Editor. JAMA 1983;249(15):2018.
22. Hopkins MP et al., Ginseng face cream and unexplained vaginal bleeding. Am J Obstet Gynecol 1988; 159(5):1,121-2.
23. Hogan RP. Hemorrhagic diathesis caused by drinking an herbal tea. JAMA 1983;249(19):2,679-80.
24. Norcross WA et al., Accidental poisoning by warfarin-contaminated herbal tea. West J Med 1993; 159(l):80-2.
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28. Zhou Q The effect of new breviscapine on the fibrinolysis and anticoagulation of human endothelial cells (Abstract). Thromb Haemost 1991:65(6):1224.
29. Wang Z et al., Effects of radix salviae miltorrhizae on antithrombotic activities of endothelial cells. Thrornb Haemost 1993:69(6):642.
30. Pansatiankul BJ et al., Dicumarol content in alcoholic herb elixirs: One of the factors at risk induced IVKD-1. Southeast Asian J Trop Med Public Health 1993;24(l):201-30.
31. Thomas 00. Anticoagulant and antifibrinolytic properties of Tanacetum cilicium. Fitoterapia 1989; 60(12):138-40.
32. Thomas 00. Anticoagulant and antifibrinolytic properties of Tanacetum corymbosum. Fitoterapia 1989; 60(3)231-3.
33. Thomas 00. Anticoagulant and antifibrinolytic properties of Tanacetum macrophyllum. Fitoterapicx 1989; 60(4):329-30.
34. Srivastava KC. Extracts from two frequently consumed spices-cumin (cuminum cyminum) and turmeric (curcuma longa) -inhibit platelet aggregation and alter eicosanoid biosynthesis in human blood platelets. Prostaglandins Leukot Essent Fatty Acids 1989; 37(l):57-64,
35. Kassler, WJ et al., The use of medicinal herbs by HIV infected patients. Arch. Intern. Med. 1991, 151:2,281-2,288.
36. Hokama, Y & Hokama, IL: In vitro inhibition of platelet aggregation with low dalton compounds from aqueous dialysates of edible fungi. Res Commun Chem Pathol Pharmacol 1981, 31:177-180.
37. Shimuzu, A et al., Isolation of an inhibitor of platelet aggregation from a fungus. Canoderma lucidum. Chem Pharm Bull. 1985, 33:3,012-3,013.
38. You, S et al., Anti-blood platelet aggregation constituents in the root of Isatis tinctoria L. Chung Yao Tung Pao. 1988, 13:31-32.
39. Ko, F et al., Inhibition of platelet thromboxane formation and phosphoinositides breakdown by osthole from Angelica pubescans. Trhromb. Haemost. 1989, 62:996-999.
40. Onitsuka, M et al., New platelet aggregation inhibitors from Tan-Shen: radix of Salvia miltiorrhiza. Chem. Pharm. Bull. 1983, 31:1,670-1,675.
41. Austin, F: Schistosoma mansoni chemoprophylaxis with dietary lapachol. Am. J. Trop. Hygien. 1974, 23:412-419.
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44. Hata, K et al., On the coumarins of the roots of Angelica polymorpha maxim (Umbelliferae). Yakugaku Zasshi. 1967, 87:464-465.
45. Hsu, H: Oriental Materia Medica. Long Beach, Calif. Oriental Healing Arts Institute, 1986.
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Aromatherapy - Essential Oils
Related Health Conditions
Anemia Menstruation Anxiety Nosebleed Blood clot Pain Bruise Pregnancy Cirrhosis Thrombocytopenia Hemophilia Ulcers Hepatitis Varicose veins Infection Vomiting
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