Sudden Infant Death Syndrome (SIDS), refers to the mysterious death of an infant during the first months of life who goes to bed apparently healthy but never wakes up.
There may be a link between the position a baby is placed in to go to sleep and the type of pillow the head is placed on. However, most babies do not smother themselves; they simply stop breathing.
Some physicians suspect a nutritional deficiency. Others point to a developing cold. Some babies have been found in time to be resuscitated. This group is at high risk for another episode and parents may place a special alarm system near the cot, which signals whenever there is a delay in the baby's breathing.
Signs & Symptoms
Usually, the only sign is that the baby stops breathing. A "near miss baby" can place a tremendous burden upon parents, straining to hear his breathing all day, every day, possibly aided by alarms or other family members.
After one (or more) babies die in the same family, the parents must also undergo interrogation from police and deal with a loss of self-confidence, as well as losing respect in the community for their ability to take care of children.
*Please refer to the respective topic for specific nutrient amounts.
Note 1: Magnesium deficiency must be resolved pre-natally in the mother, continuing during lactation.
Note 2: All amounts are in addition to those supplements having a Recommended Dietary Allowance (RDA). Due to individual needs, one must always be aware of a possible undetermined effect when taking nutritional supplements. If any disturbances from the use of a particular supplement should occur, stop its use immediately and seek the care of a qualified health care professional.
The infant diet, of course, is rather limited at first to breast or formula milk. Maternal nutrition, or formula selection (or adaptation) could play a role in this disease, especially if changes are made pre-natally.
B Complex vitamins are well-known in cases of neural tube defects and there is some indication that low minerals can be a key factor in this condition.
Over-the-counter homeopathic remedies may be single strength (of fairly weak potency e.g. 6X ) or a blend of several weaker strengths (6X, 8X, 10X).
This may comprise a single remedy, or several remedies.
Doses are administered on a 3 times daily (tid), between meals,schedule and continued for 3 days.
Liquid preparations usually use 8-10 drops per dose.
Solid preparations are usually 2 or 3 pellets per dose.
Children use 1/2 dose i.e. 1 pellet.
If there is aggravation of the symptoms, stop taking the remedy and consult a homeopath.
Murphy, R. : Homeopathic Medical Repertory. Hahneman Academy, Pagosa Springs, Colorado. 1993.
Murphy, R. : Lotus Materia Medica. Hahneman Academy, Pagosa Springs, Colorado. 1995.
Pert, J.C.: Homeopathy for the Family. The Homoeopathic Development Foundation, London. 1985 edition.
Note 1: Most deficiencies can best be resolved pre-natally in the mother, continuing during lactation. Some herbs can be soaked in formulas, or capsules may be emptied into them, or mild teas or juices can make an acceptable alternative. If necessary, sweeten with honey, or Stevia.
Note 2: The misdirected use of an herb can produce severely adverse effects, especially in combination with prescription drugs. This Herbal information is for educational purposes and is not intended as a replacement for medical advice.
Aromatherapy - Essential Oils
One theory of SIDS is that the urine released by the infant overpowers it. This may be a direct effect of the ammonia, or a combination of the ammonia and any synthetic fibers it come sinto contact with.
In this scenario, a masking odor, would be most beneficial, although the reacting materials should also be removed, or, preferably, guarded against and never installed.
Some essential oils lessen the risk of infection and may be dropped directly onto the bedding:
Eucalyptus Essence Lavender Essence Tea Tree Essence
More powerful, antiseptic aromas, which may be diffused through the air of the room, or an adjoining laundry room for the diapers, include:
Cinnamon Essence Oregano Essence (Red) Thyme Essence
This is also applicable to other sick room situations.
Related Health ConditionsAbstracts
Anonymous: Distinguishing sudden infant death syndrome from child abuse fatalities. Committee on Child Abuse and Neglect, 1993-1994. Del Med J, 1997 Jul, 69:7, 371-5.
Byard-RW et al: Sudden infant death syndrome: effect of breast and formula feeding on frontal cortex and brainstem lipid composition. J-Paediatr-Child-Health. 1995 Feb; 31(1): 14-6.
Chalmers RA et al., Mitochondrial carnitine-acylcarnitine translocase deficiency presenting as sudden neonatal death [see comments]. J Pediatr, 1997 Aug, 131:2, 220-5.
David CM: Sudden infant death syndrome: a hypothesis. Med Hypotheses, 1997 Jul, 49:1, 61-7.
Einspieler C et al., Temporal disparity between reduction of cot death and reduction of prone sleeping prevalence. Early Hum Dev, 1997 Sep 19, 49:2, 123-33.
Ford RP et al., SIDS, illness, and acute medical care. New Zealand Cot Death Study Group. Arch Dis Child, 1997 Jul, 77:1, 54-5.
Forsyth-S & Fowlie-P: Caring for the future. Modern-Midwife. 1995 Jul; 5(7): 23-6. (31 ref)
Gordon-M: Clinical update. Why breastfeeding is best for babies. Health-Visitor. 1995 May; 68(5): 203-4. (24 ref)
Kreuzer PE et al., 2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD) and congeners in infants. A toxicokinetic model of human lifetime body burden by TCDD with special emphasis on its uptake by nutrition. Arch Toxicol, 1997, 71:6, 383-400.
Lindsay-JA et al: Can superantigens trigger sudden infant death? Med-Hypotheses. 1994 Aug; 43(2): 81-5.
MacDorman MF et al., Sudden infant death syndrome and smoking in the United States and Sweden. Am J Epidemiol, 1997 Aug 1, 146:3, 249-57.
Mage DT & Donner M: A genetic basis for the sudden infant death syndrome sex ratio. Med Hypotheses, 1997 Feb, 48:2, 137-42.
Meer RR et al., Human disease associated with Clostridium perfringens enterotoxin. Rev Environ Contam Toxicol, 1997, 150:, 75-94.
Mitchell EA & Stewart AW: Gender and the sudden infant death syndrome. New Zealand Cot Death Study Group. Acta Paediatr, 1997 Aug, 86:8, 854-6.
NANN: Infant developmental care guidelines. NANN (Petaluma, CA) 1993 (14 p) (56 ref)
O'Connor RP & Persinger MA: Geophysical variables and behavior: LXXXII. A strong association between sudden infant death syndrome and increments of global geomagnetic activity--a possible support for the elimination hypothesis. Percept Mot Skills, 1997 Apr, 84:2, 395-402.
Parish AR: Sudden infant death syndrome: a proposed discovery. Med Hypotheses, 1997 Aug, 49:2, 177-9.
Reid GM & Tervit H: Sudden infant death syndrome and placental disorders: the thyroid-selenium link. Med Hypotheses, 1997 Apr, 48:4, 317-24.
Rinaldo P et al., Sudden neonatal death in carnitine transporter deficiency [see comments]. J Pediatr, 1997 Aug, 131:2, 304-5.
Schluter PJ et al., Housing and sudden infant death syndrome. The New Zealand Cot Death Study Group. N Z Med J, 1997 Jul 11, 110:1047, 243-6.
Sivan Y et al., Home monitoring for infants at high risk for the sudden infant death syndrome. Isr J Med Sci, 1997 Jan, 33:1, 45-9.
Thomas-DB: Cleft palate, mortality and morbidity in infants of substance abusing mothers. J-Paediatr-Child-Health. 1995 Oct; 31(5): 457-60.
Vandenplas Y et al., The relation between gastro-oesophageal reflux, sleeping-position and sudden infant death and its impact on positional therapy. Eur J Pediatr, 1997 Feb, 156:2, 104-6.
Wells JC: Can risk factors for over-heating explain epidemiological features of sudden infant death syndrome? Med Hypotheses, 1997 Feb, 48:2, 103-6.
Woodruff TJ et al., The relationship between selected causes of postneonatal infant mortality and particulate air pollution in the United States. Environ Health Perspect, 1997 Jun, 105:6, 608-12.
Woolridge-M: The Baby Friendly Hospital Initiative UK Modern-Midwife. 1994 May; 4(5): 32-3. (15 ref)
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